Kungenzeka ukwazi ukuthi isiguli sinomsebenzi ongavamile wokugaya ngaphambi kokuhlinzwa, ukuvimbela ngempumelelo izimo ezingalindelekile njengokuphuma kwegazi okungapheli ngesikhathi nangemva kokuhlinzwa, ukuze kutholakale umphumela omuhle kakhulu wokuhlinzwa.
Umsebenzi womzimba wokwelula igazi ufezwa yisenzo esihlangene sama-platelet, uhlelo lokujiya kwegazi, uhlelo lwe-fibrinolytic kanye nohlelo lwe-vascular endothelial. Esikhathini esidlule, besisebenzisa isikhathi sokuphuma kwegazi njengesivivinyo sokuhlola amaphutha okusebenza kwe-hemostatic, kodwa ngenxa yokulinganisa kwaso okuphansi, ukuzwela okubi, kanye nokungakwazi ukubonakalisa okuqukethwe kanye nomsebenzi wezinto ezijiya igazi, sithathelwe indawo ukuhlolwa komsebenzi wokujiya kwegazi. Ukuhlolwa komsebenzi wokujiya kwegazi kuhlanganisa kakhulu isikhathi se-plasma prothrombin (PT) kanye nomsebenzi we-PT obalwa kusukela ku-PT, isilinganiso samazwe ngamazwe esijwayelekile (INR), i-fibrinogen (FIB), isikhathi se-activated partial thromboplastin (APTT) kanye nesikhathi se-plasma thrombin (TT).
I-PT ikhombisa kakhulu umsebenzi wohlelo lokujiya kwangaphandle. I-PT ende ibonakala kakhulu ekunciphiseni kwe-congenital coagulation factor II, V, VII, kanye ne-X, ukuntuleka kwe-fibrinogen, ukuntuleka kwe-acquired coagulation factor (i-DIC, i-primary hyperfibrinolysis, i-obstructive jaundice, ukuntuleka kwe-vitamin K, kanye nezinto ezilwa nokuvuvukala kwegazi. Ukufinyela kwe-PT kubonakala kakhulu ekwandeni kwe-congenital coagulation factor V, i-DIC yokuqala, izifo ze-thrombotic, izithibela-nzalo zomlomo, njll.; ukuqapha i-PT kungasetshenziswa njengokuqapha imithi yokwelapha ye-anticoagulant yomlomo.
I-APTT iyisivivinyo sokuhlola esithembeke kakhulu sokuntuleka kwe-endogenous coagulation factor. I-APTT ende ibonakala kakhulu kwi-hemophilia, i-DIC, isifo sesibindi, kanye nokumpontshelwa igazi eligciniwe okukhulu. I-APTT emfushane ibonakala kakhulu kwi-DIC, isimo se-prothrombotic, kanye nezifo ze-thrombotic. I-APTT ingasetshenziswa njengesibonakaliso sokuqapha ukwelashwa kwe-heparin.
Ukwelulwa kwe-TT kubonakala ku-hypofibrinogenemia kanye ne-dysfibrinogenemia, ukwanda kwe-FDP egazini (DIC), kanye nokuba khona kwe-heparin kanye nezinto ze-heparinoid egazini (isb., ngesikhathi sokwelashwa nge-heparin, i-SLE, isifo sesibindi, njll.).
Kwakukhona isiguli esiphuthumayo esathola izivivinyo zelebhu ngaphambi kokuhlinzwa, futhi imiphumela yokuhlolwa kokuqina kwegazi yayiyi-PT ne-APTT ende, futhi i-DIC yayisolwa esigulini. Ngaphansi kwesincomo selebhu, isiguli senza uchungechunge lwezivivinyo ze-DIC futhi imiphumela yayimihle. Azikho izimpawu ezisobala ze-DIC. Uma isiguli singenaso isivivinyo sokuqina kwegazi, kanye nokuhlinzwa okuqondile, imiphumela izoba mibi kakhulu. Izinkinga eziningi ezinjalo zingatholakala esivivinyweni somsebenzi wokuqina kwegazi, esithenge isikhathi esengeziwe sokuthola nokwelapha izifo emtholampilo. Ukuhlolwa kochungechunge lokuqina kwegazi kuyisilingo esibalulekile selebhu somsebenzi wokuqina kwegazi weziguli, esingabona umsebenzi wokuqina kwegazi ongavamile ezigulini ngaphambi kokuhlinzwa, futhi kufanele sinakwe ngokwanele.
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