Cov kab mob hemorrhagic uas tuaj yeem faib ua pawg yog dab tsi?


Tus Sau: Succeeder   

Muaj ntau hom kab mob ntshav, uas feem ntau yog muab faib ua pawg raws li lawv qhov etiology thiab pathogenesis. Nws tuaj yeem muab faib ua vascular, platelet, coagulation factor abnormalities, thiab lwm yam.
1. Cov hlab ntsha:
(1) Los ntawm noob caj noob ces: cov kab mob telangiectasia uas los ntawm noob caj noob ces, cov kab mob ntshav hauv cov hlab ntsha, thiab cov nqaij mos uas txhawb nqa tsis zoo nyob ib puag ncig cov hlab ntsha;
(2) Tau txais: kev puas tsuaj rau phab ntsa ntawm cov hlab ntsha los ntawm kev ua xua rau cov ntshav, cov ntshav yooj yim, cov ntshav los ntawm tshuaj, cov ntshav uas cuam tshuam nrog hnub nyoog, cov ntshav tiv thaiv kab mob autoimmune, kev kis kab mob, cov yam ntxwv ntawm kev ua haujlwm ntawm lub cev, cov yam ntxwv tshuaj lom neeg, cov yam ntxwv kho tshuab, thiab lwm yam.

2. Cov khoom ntawm cov platelet:
(1) Thrombocytopenia: kev tiv thaiv kab mob thrombocytopenia, tshuaj ua rau thrombocytopenia, aplastic anemia, qog infiltration, leukemia, kab mob tiv thaiv kab mob, DIC, splenic hyperfunction, thrombotic thrombocytopenic purpura, thiab lwm yam;
(2) Thrombocytosis: Primary thrombocytosis, true polycythemia, splenectomy, o, inflammatory platelet dysfunction, thrombocytopenia, giant platelet syndrome, kab mob siab, thiab platelet dysfunction los ntawm uremia.

3. Cov yam ntxwv tsis sib xws ntawm cov ntshav khov:
(1) Cov teeb meem coagulation factor uas los ntawm noob caj noob ces: hemophilia A, hemophilia B, FXI, FV, FXI, FVII, FVIII, tsis muaj peev xwm, tsis muaj fibrinogen txij thaum yug los (tsis muaj), tsis muaj prothrombin, thiab tsis muaj complex coagulation factor;
(2) Cov teeb meem coagulation factor uas tau txais: kab mob siab, tsis muaj vitamin K, mob leukemia mob hnyav, lymphoma, kab mob connective tissue, thiab lwm yam.

4. Kev ua kom cov ntshav khov ntau dhau:
(1) Thawj Zaug: Kev tsis txaus cov tshuaj fibrinolytic inhibitors lossis kev ua haujlwm plasminogen ntau ntxiv tuaj yeem ua rau hyperfibrinolysis yooj yim hauv cov kab mob siab hnyav, qog nqaij hlav, phais, thiab raug mob;
(2) Tau txais: pom tseeb hauv thrombosis, DIC, thiab kab mob siab hnyav (theem ob)

Kev nce ntxiv ntawm cov tshuaj uas ncig mus los, cov tshuaj uas tau txais los ntawm cov tshuaj inhibitors xws li F VIII, FX, F XI, thiab F XII, cov kab mob autoimmune, cov qog nqaij hlav phem, cov tshuaj heparin zoo li anticoagulants ntau ntxiv, thiab lupus anticoagulants.

Siv: [1] Xia Wei, Chen Tingmei. Cov Txuj Ci Ntsuas Ntshav Hauv Tsev Kho Mob. Tsab Ntawv Thib 6 [M]. Beijing. Tsev Luam Tawm Txog Kev Noj Qab Haus Huv ntawm Tib Neeg. 2015

Beijing SUCCEEDER https://www.succeeder.com/ ua ib lub npe lag luam hauv Suav teb Diagnostic kev lag luam ntawm Thrombosis thiab Hemostasis, SUCCEEDER tau muaj kev paub txog pab pawg R & D, Kev Tsim Khoom, Kev Lag Luam Muag Khoom thiab Kev Pabcuam Muab cov tshuaj ntsuas coagulation thiab reagents, cov ntshav rheology analyzers, ESR thiab HCT analyzers, platelet aggregation analyzers nrog ISO13485, CE Daim Ntawv Pov Thawj thiab FDA teev npe.